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WHEN THE HGH REVOLUTION BEGAN


Written by Dr. Welsh, Published on 28 August 2012

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The HGH Growth Hormone revolution began with the Rudman study, it is the birth of the modern Real Injectable Prescription Real Injectable Prescription HGH Growth Hormone Growth Hormone  revolution. In the late 1980's, 1989 to be exact, a well respected endocrinologist from the University of Wisconsin by the name of Donald Rudman, conducted an experiment where all the participants were given medical Real Injectable Prescription Real Injectable Prescription HGH Growth Hormone Growth Hormone injection for about six months. The participants were specifically told not to change anything in their schedule or lifestyles. After the six month period had elapsed most of the participants noticed some significant changes in their health.
Some of the results reported were: weight reduction and weight loss, younger, healthier, thicker skin, better energy and sexual stamina, stronger healthier hair, less wrinkles, supple skin, and generally an overall better well being were among the immediately noticeable effects of Real Injectable Prescription Real Injectable Prescription HGH Growth Hormone Growth Hormone injections.  Many of the subjects of this HGH Growth Hormone experiment felt years younger. Still, the proof from Rudman’s study was not enough to change the mind of the medical establishment that still considers it a type of quackery. Hundreds of studies have been conducted since 1989 on Real Injectable Prescription Real Injectable Prescription HGH Growth Hormone Growth Hormone and more than not, the studies show Real Injectable Prescription Real Injectable Prescription HGH Growth Hormone Growth Hormone has many anti-aging properties. 

THEORIES ON AGING


Written by Dr. Welsh, Published on 15 March 2014

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Theories on Aging

Aging is a complex process which has a variety of working parts. Medical scientists and biological researchers continue to study aging in order to give humanity a more complete understanding of the mechanisms by which it occurs, and they also try to learn ways to slow down or even reverse some aspects of the aging process.


There are three primary mechanisms that control how we age: Genetic Aging, Biochemical Aging, and Physiological Aging.

Genetic Aging

One of the three mechanisms by which human beings age is genetic. There are many forms of genetic aging, but all revolve around the idea that genetic breakdown causes the body to age over time. As we grow older, the genes become more prone to mutate. These mutations can cause a variety of issues to occur, one of which is cancer.

If a gene alters in a way that encourages endless replication, this significantly increases mortality risk, for example. Genetic mutations can also lead to other, less severe, genetic issues which simply impede the body's ability to perform the functions necessary to maintain a healthy and optimally functional body.

Small formations on the ends of our genes known as Telomeres also have an impact on aging. Telomeres are sections of junk genes at the end of our chromosomes which keep the active genetic code intact. As we age, these Telomeres become shorter and shorter, increasing the risk of genetic malfunction and mutation.

Finally, there are certain genes that only seem to express themselves later in life or when certain negative pressures are applied. And these negative pressures cause the aging process to occur more quickly.

Biochemical Aging

Biochemical Aging is a precursor of many forms of Physiological Aging. Biochemical Aging refers to intracellular and extracellular processes which contribute to aging in some way. In some cases, this is the result of natural chemical processes which cause our body to slowly break down over time, such as the inevitable oxidization which occurs as a natural side-effect of the chemical processes which keep us alive.

Other symptoms of biological aging are the result of changes in the way that our body functions, often for reasons that we are just beginning to understand. The way that our bodies produce Human Growth Hormone and Testosterone are good examples of this form of Biochemical Aging.

When we are young, our bodies produce ample levels of these hormones, and it encourages growth during adolescence and supports good health during early adulthood. Around the age of thirty, however, these hormones start to decline because the brain sends fewer and fewer signals for them. Also, hormones which control stress are produced in lower concentrations with age, which can increase the influence of psychological and physiological stress, which can cause other issues related to aging to become much more dangerous and severe.

Physiological Aging

Physiological aging refers to the physical changes that take place as a result of aging. Our teeth represent one of the most noticeable markers of physiological aging. Our teeth grow into place after our baby teeth fall out when we are young. After your teeth fully develop, your body has no mechanism to rebuild them and they slowly degrade over time, at a rate dependent upon how well you take care of them.

Graying hair is another type of physiological aging. Hair color is actually produced by tiny cellular organs known as Melanocytes. The color of your hair is dependent upon the type of pigment that these cells create, as well as the quantity and concentration at which they are produced. The more melanin that the Melanocytes produce, the darker your hair will be.

People get gray hair because the older that they get, the less pigment that Melanocytes secrete over time. There are actually scientific studies being performed regarding how to influence these cells to continue producing pigment later in life, potentially allowing aging men and women to naturally maintain their natural hair color.

Other Physiological aging changes are more than cosmetic, however. There are certain processes that occur regardless of how you take care of yourself as you grow older. Your baseline blood pressure will slowly go up over time, and your heart muscles will slowly harden, a condition known as atherosclerosis. Many physiological signs of aging can be alleviated with medication or a healthy lifestyle, but the general trends will continue over time without particular forms of treatment (many of which don't exist today).

How We Age Does Not Explain Why We Age

These three concepts, Physiological Aging, Biochemical Aging, and Genetic Aging, only serve to explain the processes by which we age. These processes do not answer the ultimate question of Why We Age, however. There are a number of theories associated with answering the question of why we age, but all of these theories belong to two distinct classes: Program Theory and Error Theory.

Program Theory assumes that aging occurs for evolutionary reasons, as a means to improve the effective viability of the species. There are a number of means by which this can be true, and it assumes that from the day that we are born, we are designed to progress down a set path from birth to death which maximizes the long term survival of the species. These programmed theories can be considered like a series of stages of life that eventually leads to death.

Error Theory, on the other hand, assumes that we are not genetically designed to self-destruct, but it is the result of a potential multitude of malfunctions which slowly reduce the viability of an organism until it eventually dies. There are a wide variety of forces that pressure an organism to break down and reach the end of its lifespan. It is incredibly important to note that the reality is likely much messier than a single theory, and there are inevitably a multitude of pressures which lead to aging, which are likely related to hypotheses of both Program Theory and Error Theory.

Below is a collection of some of the theories associated with Why We Age:

Program Theories of Aging

Programmed Senescence

This theory behind aging centers around the idea that we are programmed to age from a genetic level. It assumes that there are certain genes that turn on at a certain stage of the lifespan which change the way that our body works at a cellular level. This creates a cascade of change which causes our bodies to slowly deteriorate over time.

There are a number of cellular processes in the human body that involve programmed cell death, which is also known as apoptosis. Advanced theories regarding Programmed Senescence hypothesize that the genetic code of all animal species induces aging as a form of organism-wide apoptosis which makes way for the newer, younger generations.

Endocrine Theory

This Programmed Aging Theory hypothesizes that the endocrine system has the ability to control the rate at which an organism ages based off of both a natural biological clock as well as outside influences which may induce or postpone aging. This theory is also known as the Hormone Theory of Aging.

There are many insects, for example, that have the ability to live much longer lives if the conditions are not ideal for reproduction and procreation. If it is excessively cold, there is not enough food, or there are not enough partners, many insect species will release hormones which halt or slow down aging until conditions are more ripe to create offspring.

In human beings and other mammals, Growth Hormone and Sex Hormone production declines around midlife until the end of the lifespan. There is powerful evidence that these hormones have a positive impact on health and longevity, and there are those that argue that the body reduces production of these Anti-Aging Hormones over time so that the older generation can make way for the new generation.

Immunological Theory

This aging theory makes the hypothesis that immune systems weaken over time as a result of evolutionary pressures to encourage the organism to eventually become ill and die. This theory is also known as Immunosenescence. When we are young we are incredibly resilient to disease, but the older that we get, the less able our bodies are to fend off illness.

There is some evidence that an organism's ability to fight disease drops over time without regard to external factors, which leads some to suggest that this is an evolutionary strategy to encourage the young and healthy to acquire dominance over time. This change isn't exclusively the result of exterior factors, but as a result of changes which are deeply embedded in our genetic code.

Error Theories of Aging

Wear and Tear

This theory is quite straight forward. Our bodies grow and develop through childhood and adolescence, but after our bodies reach a point of peak performance, certain parts of our body start to age and decline in function because they do not have the capacity to heal themselves or have a limited ability to heal themselves.

One example of wear and tear is the scarring deterioration of the skin. When you receive a cut, your body has the ability to seal itself up and maintain a closed system, but if scars don't heal properly they will remain on the skin the rest of your life, because the body does not have a means to make the marks associated with these old wounds disappear once they are fully healed.

Another example of wear and tear involves the liver. The liver is an organ that has a limited ability to heal itself. Liver damage can result from a number of different lifestyle choices, including alcoholism, obesity, and drug abuse. The liver expends a lot of energy filtering impurities out of the body, and if too much is consumed for too long, the liver starts to become overwhelmed by fat tissue and cirrhosis.

Rate of Living

This form of aging is based on the hypothesis that, regardless of any evolutionary benefit, animal organisms have a lifespan which is highly correlated with their metabolism throughout their lives.

For example, rabbits and other small animals with very fast heartbeats tend to have very short lifespans. Animals like turtles, whales, and elephants have slow metabolisms which are associated with very slow heart beats, and they tend to have very long lifespans.

Although metabolism does seem to play a certain correlative factor with regard to the average lifespan of various species, it is not a hard rule, and evidence suggests that it does not apply effectively to humans. Low Metabolism in humans is associated with a shorter lifespan, and is correlated with health issues such as obesity and cardiovascular disease.

Crosslinking

Crosslinking is a theory of aging that assumes that the older that we get, the more links that the proteins in our body make, causing them to no longer function as effectively. One example of crosslinking which leads to physical aging is skin elasticity.

When we are born, our skin is soft and returns to position quickly if pulled or compressed. This is because the collagen in our skin is optimally linked. The older that we get, the more crosslinkages that are made among the collagen of our skin, which causes the skin to tighten and become less optimized. These links eventually start to cause skin cells to function ineffectively or even die off because the protein links are so tight that nutrients are not delivered efficiently.

Crosslinking is also one reason why the immune system loses its function over time. The body loses its ability to successfully process glucose from the blood. This causes blood sugar to increase which has a toxic effect on the cardiovascular system. Sugars in the blood react with protein, increasing the prevalence of free radicals which can do major damage to the body over time.

Free Radicals

Free Radicals are the result of the very chemical processes of the human body which sustain us. As our bodies react with oxygen to create energy, they release spare atoms which travel haphazardly through the body causing chemical reactions and reaction cascades that can do harm to the body.

One of the body's primary lines of defense against Free Radicals are nutrients known as Antioxidants. The human body has the ability to produce some forms of antioxidant naturally, but others must be consumed in the diet. Omega-Three Fatty Acids, L-Carnitine, and Vitamin-C are some examples of antioxidants.

Free Radicals cause aging because the incremental damage caused by these chemical reactions slowly degrades the body, causing organs and cells to lose their ability to properly function and this slowly leads to declining health and vastly increased mortality risk.

Error Catastrophe

This form of Error Theory is associated with the way that our bodies utilize proteins. As a result of various other interferences, the cellular organs which generate necessary proteins in our body can malfunction, causing them to produce damaged proteins which can negatively impact health and normal function.

One of the most well-known forms of Error Catastrophe is actually Alzheimer's Disease. Alzheimer's Disease is caused when our brains become overloaded with prions, which are proteins which the body has folded improperly. These prions have the ability to convert other healthy proteins into the same form, spreading damage throughout the brain, deteriorating normal function over time until eventually inevitably leading to death.

Error Catastrophe also refers to the point at which mutations in proteins or genes reach a point at which the cell can no longer survive and propagate under normal circumstances. Eventually, the organism dies because cellular function breaks down at organ level and finally at a universal level.

Somatic Mutation

This form of Error Theory is related to Error Catastrophe, but is explicitly related to genetic function. A number of different factors can eventually cause genetic function to alter, which causes aging and eventually can lead to death.

Cancer is often the result of Somatic Mutation. A gene can mutate, causing it to reproduce indefinitely. This genetic mutation alters the healthy function of the body, and over time, the cancer can spread to other areas of the body and flood the entire organism with replicating cancer cells, which will inevitably lead to death. Cancers can usually be treated effectively if caught early, but if allowed to spread, they often become almost universally lethal.

Somatic Mutations can occur at any time from conception to the end of the lifespan. Sometimes, these mutations lie dormant for many years, perhaps even through the entire lifespan, but other times, they eventually lead to cancer or other physiological malfunctions.


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One Third of Type-Two Diabetics Suffer from Testosterone Deficiency

A new study shows that young male patients with Type-Two Diabetes have a significantly high risk of developing Testosterone Deficiency at an early age. This study shows that around one in three patients between the ages of 18 and 35 suffer from Clinically Low Levels of Testosterone.

Diabetes-Testosterone Study Information

The primary investigator in this study was Dr. Paresh Dandona. He and his associates performed their research at the New York State University of Buffalo. This study compared 62 diabetic patients. 24 of the men had been previously diagnosed with Type-Two Diabetes and 28 of the participants were males with Type One Diabetes.

Blood testing revealed that patients with Type-Two Diabetes had much lower endogenous Testosterone Levels than the male patients with Type One Diabetes. 33% of the subjects with Type-Two Diabetes were found to have clinically low levels of Testosterone, and 58% of this group had Testosterone levels which were abnormally low in comparison to healthy patients. Type-One Diabetes patients did not suffer from this clinical medical issue, however. Only 8% of Type-One Diabetics were found to have Testosterone levels which were at all abnormal.

Testosterone Deficiency Linked to FSH Deficiency and Luteinizing Hormone Deficiency

Patients in this study that were found to have low levels of Testosterone were also shown to have medically low levels of Follicle-Stimulating Hormone and Luteinizing Hormone. As a result of this, these patients clinically suffered from a disorder known as Hypogonadotrophic Hypogonadism. This disorder greatly enhances the risk of a number of medical disorders if left untreated, including cardiovascular disease and osteoporosis. Other symptoms of Hypogonadotrophic Hypogonadism include infertility, impotence, and lack of libido.

Type-Two Diabetics are High Risk Patients for Low Testosterone

In the end, the researchers came to the conclusion that young patients suffering from Type-Two Diabetes are an incredibly at-risk subset of the population for suffering from Low-T, even in their twenties. Both Free Testosterone levels and Total Testosterone levels are significantly affected. As a result of this deficiency in combination with deficiencies of Luteinizing Hormone and Follicle-Stimulating Hormone, these patients are at a high risk of a number of cardiovascular and sexual medical conditions and need to be treated with Testosterone Hormone Replacement Therapy in order to minimize health risks and help them live a healthier and more fertile life.

Facts About Testosterone Deficiency

Testosterone Deficiency is a very common symptom of both diabetes and obesity. Around one third of males with Type-Two Diabetes will also suffer from the symptoms of Testosterone Deficiency as a result. The clinical term for Low-T is known as Hypogonadism.

Symptoms of Low-T

There are a number of symptoms related to Low Testosterone. Among these symptoms are:

Decreased sex drive Low Testosterone reduces sexual desire in males, leading to bedroom dissatisfaction and contributing to rocky relationships

Depression Testosterone plays a significant role in male mood stabilization. Males who suffer from Hypogonadism are more likely to experience feelings of sadness and depression that they are unable to shake.

Fatigue and exhaustion Testosterone is a vital component of male metabolism. Although Human Growth Hormone directly causes enhanced metabolism, Low Testosterone causes problems with muscle strength and cardiovascular stamina.

Bone health deterioration Patients who suffer with Low-T are at an increased risk of bone disorders such as osteoporosis. Loss of Bone Mineral Density can even make you grow shorter over time!

Reduced strength Testosterone is directly responsible for the differences in muscle strength and muscle mass that separate males and females. Testosterone has the ability to amplify the effects of weight training and muscle building. Men who suffer from Low-Testosterone lose their ability to generate and sustain additional muscle.

Decreased erectile sensitivity Healthy Testosterone Levels are necessary for men to experience optimal pleasure in sexuality. Patients with Low-T often complain the sex just doesn't feel like it used to, which puts a damper on sex drive and libido.

Fertility Testosterone is an important factor in the production of healthy sperm. Individuals with low levels of Testosterone have lower sperm counts than those who have healthy Testosterone levels. In addition to this, normal Testosterone levels are vital for healthy and fully functional sperm.

Smaller testicles One of the most obvious visible symptoms of Testosterone Deficiency is testicular shrinkage. Without sufficient levels of Testosterone, the Testicles are not as healthy or functional, and one of the ways in which this can be seen is through testicular size and shape.

Reduced sexual ability Men with Low Testosterone even complain that their disorder prevents them from even engaging in satisfying sexual activity. Testosterone is a big part of what allows a man to get turned on, and when Testosterone levels in the body are insufficient, the physiological process which produces a firm, hard erection is hindered.

Increased inflammation Low Testosterone is correlated with increased levels of cortisol, which is the human body's central agent of inflammation, which can cause pain as well as slow the body's natural healing processes.

Low-T Exacerbates Symptoms of Diabetes

Testosterone Deficiency has been shown to make the symptoms of diabetes worse. In addition to this, suffering from Low Testosterone while also dealing with Diabetes can increase the risk of many heart complications such as stroke, cardiovascular disease, and high cholesterol.

Dr. Paresh Dandona and Dr. Sandeep Dhindsa have dedicated their careers to the study of Diabetes and have found a number of critical links between Type-Two Diabetes and Testosterone Deficiency.

Type-Two Diabetes Hastens Declining Testosterone Levels

Testosterone levels naturally decline over time beginning in the late twenties, and this decline continues throughout the lifespan. In otherwise healthy individuals, this decline occurs at a rate of between one or two percent each year, similar to the rate at which Human Growth Hormone levels decline. There are a number of medical conditions which increase the rate at which Testosterone Decline occurs, and there are other disorders which greatly reduce Testosterone Levels very quickly.

Type-Two Diabetes significantly depresses natural Testosterone levels, leading to a number of life changing and potentially dangerous side-effects as a result. Studies like the one we mentioned at the beginning of this article have led endocrine scientists to the conclusion that four out of every ten men who suffer from obesity will have clinically low levels of Testosterone in comparison to males of the same age that are not overweight. When diabetes is thrown into the mix as well, the resulting difference is even more profound. Low-Testosterone levels plague fifty percent of obese males clinically diagnosed with diabetes.

Type-Two Diabetics Suffer Primarily from Secondary Hypogonadism

One may come to the conclusion that the primary correlation here would be between weight and testosterone level, but this is not necessarily the case. Although men that are both obese and diabetic are most at risk of Low Testosterone Levels, there is ample evidence that Type-Two Diabetes significantly reduces Testosterone Levels even in patients who do not suffer from weight issues. This seems to suggest that Type-Two Diabetes seriously inhibits the human body's ability to produce Testosterone is a direct physiological way.

Type-Two Diabetes Equivalent to Twenty Pounds of Fat

Dr. Dhindsa, one of the primary investigators of the study discussed at the beginning of this article, says that Diabetes has an effect on Testosterone Levels that can be correlated with weight gain. He says that those that suffer from Type-Two Diabetes experience a decline in Testosterone levels that is the equivalent of gaining twenty pounds of body fat.

Another study produced by Dr. Dhindsa shows that endogenous Testosterone production declines at a rapid and non-linear rate dependent upon Body Mass Index. The more that an individual weighs, the more likely they are going to suffer from an acute deficiency of Testosterone.

Largest Testosterone-Diabetes Study Ever Conducted

Dr. Dandona and Dr. Dhindsa have been responsible for the largest study regarding Testosterone, Diabetes, and Obesity ever conducted. Before these two doctors began their study, most research regarding Testosterone Deficiency was only concerned with patients who suffered from obesity, or patients who suffered from Diabetes. These two doctors focused on how the two diseases interact and amplify the symptoms of Testosterone Deficiency. In their largest study, they utilized data from the 2003-2004 Hypogonadism in Males study. This clinical study involved over 1800 males from 95 medical clinics. The study was underwritten by the company Solvay Pharmaceuticals.

The scientists recommend that males with diabetes and obesity keep a close eye on their Testosterone Levels. Healthy Testosterone levels are an important part of living a healthy life, and Low-Testosterone can increase the dangers of both diabetes and obesity, while also preventing patients from being able to successfully manage their disease. Dr. Dhindsa's primary goal as a medical researcher is to improve treatment options for male patients who suffer from Hypogonadism and diabetes. Dr. Dandona is primarily interested in the effects and causes of Low Testosterone in younger men who for all intents and purposes should be participating in the most reproductive period of their lives.

Questions about Testosterone Deficiency

What is Follicle-Stimulating Hormone?

Follicle-Stimulating Hormone (often abbreviated FSH) is a member of a group of hormones known as Gonadotrophins. Gonadotrophins are hormones which are responsible for the stimulation of the sexual organs which are vital for healthy sexual function. These hormones are derivatives of Testosterone and Estrogen which promote the normal function of the testes and the ovaries. FSH is both manufactured and secreted by the pituitary.

In females, FSH increases the formation of egg follicles while also stimulating the ovulation process. In males, FSH primarily stimulates a group of cells known as Sertoli cells. These cells are responsible for the healthy development of sperm cells. Low Levels of FSH in adults have no real negative consequences outside of fertility issues, but monitoring FSH levels helps doctors identify the exact source of Testosterone Deficiency. In young boys and girls experiencing puberty, however, low levels of FSH can hinder the proper development of the testes and ovaries, significantly affecting the development of secondary sexual characteristics.

What is Luteinizing Hormone?

Although Follicle-Stimulating Hormone and Luteinizing Hormone are both Gonadotrophins, a deficiency of Luteinizing Hormone is much more harmful to human physiology. Luteinizing hormone is sometimes referred to as Lutropin. This hormone is produced by the pituitary gland by formations known as Gonadotroph Cells. These cells are located in the anterior portion of the pituitary.

In women, Luteinizing Hormone is the base trigger mechanism for ovulation. In males, the hormone is responsible for stimulating the production of Testosterone by triggering the function of Leydig cells. In males, a deficiency of Luteinizing hormone can contribute to a number of negative symptoms, including Testosterone Deficiency and the symptoms inherent with that Deficiency.

A deficiency in Luteinizing Hormone during puberty can delay and limit the natural changes that occur. A Testosterone Deficiency combined with a deficiency of FSH and Luteinizing Hormone is known as Idiopathic Hypogonadotrophic Hypogonadism.

What is Hypogonadism?

Hypogonadism is the blanket term for any disorder which prevents the human body from optimally producing sex hormones such as Testosterone, Estrogen, and Progesterone. Adult-onset Testosterone Deficiency is a form of Hypogonadism which generally results from the slow and steady decreased hormone production of the testes. Hypogonadism as a result of Obesity and Type-Two Diabetes is the result of the conversion of existing Testosterone into Estrogen and Cortisol as well as reduced input from the hypothalamus and pituitary gland.

What is Hypogonadotrophic Hypogonadism?

Hypogonadotrophic Hypogonadism is a form of gonadal deficiency that is the result of an insufficiency of the pituitary gland or the hypothalamus. This range of disorders is also referred to as Secondary Hypogonadism. Primary Hypogonadism is the result of a hormonal deficiency of the sex organs. Adult-onset, age related Testosterone Deficiency is generally related to Primary Hypogonadism. Testosterone Deficiency resulting from Type-Two Diabetes is mostly the result of Secondary Hypogonadism, which is why the disorder is very powerful and not related to normal testosterone decline as a result of aging.

There are a few important steps to normal testicular function. The hypothalamus releases a hormone known as Gonadotropin-Releasing Hormone, abbreviated GnRH. GnRH then flows from the hypothalamus to the anterior pituitary gland. After stimulation, the pituitary gland secretes and releases the hormones FSH and Luteinizing Hormone. These hormones then move through the blood stream down to the male and female sex organs where they stimulate the normal production of sex hormones such as Testosterone and Estrogen as well as other important hormones which control sexual function. If there is a hormonal imbalance at any point between the hypothalamus and the sex organs, this has the potential to greatly alter normal hormone production which can be detrimental to human health.

Although there are some forms of extreme or total Hypogonadotrophic Hypogonadism, Diabetes and obesity produce a moderate form of Hypogonadotrophic Hypogonadism which causes a number of detrimental symptoms which are very similar to Age-Related Testosterone Deficiency as a result of Andropause. Most individuals will not experience the negative effects of Testosterone Deficiency until their thirties and forties, but patients who suffer from Hypogonadotrophic Hypogonadism often experience hormonal issues from birth. Many others, as is the case with those suffering from Type-Two Diabetes, begin to experience the symptoms of Testosterone Deficiency in their early twenties.

What is the importance of these Testosterone Deficiency Studies?

Dr. Dandona's research study sheds a significant amount of light on how Testosterone Deficiency, Obesity, and Diabetes interact. The pathology of Testosterone Deficiency in Type-Two Diabetes does not seem to be related to an insufficiency of the testes, but to issues resulting from hormonal balance resulting from the pituitary gland and the hypothalamus. This causes symptoms of hormonal deficiency which normally do not begin to occur until patients are in their thirties and forties to appear while patients are in their twenties.

Fatigue and exhaustion prevent patients from efficiently engaging in exercise, and changes in muscular metabolism prevent them from building muscle as a result of exercise. Low Testosterone also encourages higher levels cortisol and estrogen, which are incredibly detrimental to male patients and reduce cardiovascular health significantly. Obesity complicates issues with Low Testosterone and Type-Two Diabetes by increasing the rate at which Testosterone is converted into estrogen by the fat cells. Adipose fat tissue naturally has the ability to convert Testosterone into Estrogen and Estrogen-related hormones. The more fat tissue present on the body, the less of an impact endogenous Testosterone produces on the human body.

Scientific evidence shows that Type-Two Diabetes and Obesity are intricately connected in a number of ways, although not all diabetics are obese and not all obese individuals develop diabetes. Diabetes reduces the effectiveness of insulin to process blood sugar which makes it harder to maintain a healthy body weight and deteriorates physical health in a numerous ways. Dr. Dandona's study shows that both obesity and Type-Two Diabetes impact Testosterone negatively via their own unique yet interconnected pathways.

Also, the study shows that Testosterone Hormone Replacement Therapy can be a highly beneficial treatment that alleviates some of the effects of Diabetes while also improving metabolism, boosting energy levels, and encouraging weight loss in order to help obese patients lose weight and improve their BMI.

If you suffer from Type-Two Diabetes we encourage you to monitor your Testosterone Levels closely. Although you may be in control of your insulin levels, you may be allowing Low Testosterone to decrease the quality of your life in significant ways. To learn more about Testosterone Hormone Replacement Therapy, feel free to contact the Conscious Evolution Institute with any questions you may have!


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Testosterone Therapy Benefits

Correctly performed testosterone therapy can be your ticket to health.

Three sided solution: Testosterone + HCG + Arimidex

If your doctor only prescribes testosterone by itself, you will probably have a rough ride. The tendency is for you to feel great the first couple months, while you increase testosterone levels, followed by a slow deterioration, once your estrogen creeps up.

High estrogen negates a lot of the positives from testosterone therapy, resulting in the same symptoms of low testosterone you had in the first place!

The solution is to add a drug called Arimidex. It's called an aromatase inhibitor, which essentially blocks the conversion of testosterone to estrogen. It has the effect of increasing testosterone levels, while keeping your estrogen low.

Once you have your testosterone and estrogen solved, it's time to stop the next inevitable decline? Shrinking testicles.

This is where HCG (human chorionic gonadotropin) comes in. It prevents both infertility and testicle shrinkage. Your testicles shrink because your body thinks it doesn't need to make testosterone anymore.

For some, small testicles may seem like just a cosmetic problem. But HGC does more than increase testicle size, it also increases adrenal function, which can have positive effects on well-being, libido, and energy.





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