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Does Growth Hormone Have Anti-Aging Properties?

It is quite true that we are all going to get old. As a matter of fact, the generation of baby boomers is quickly approaching their mid-fifties, and the number of elderly people in America will continue to increase for another generation. Why then, do we have to resign to growing old in the same way that previous generations were forced to in the ages before us? Must we be weak, frail, fatigued, depressed? Living in fear to take a walk in the park for fear that we may fall?

It is a truism that youth is wasted on those who are young. If such a saying is true, why should the opposite statement also be true? Why must wealth and experience be wasted upon those that are old? Should not older men and women be able to retain their enthusiasm and energy in a grand effort to enjoy their autumn years?

We do not have to take age lying down! We here at the Conscious Evolution Medical Institute have learned that old age is largely a state of mind that has been induced by hormonal deficiencies. It is a state in which the hormones we so enjoyed in our youth have become less bountiful. It makes logical sense that with hrt hormone replacement therapy many of these symptoms can be alleviated or reversed.

One of the most impressive hormones that can be regained through Bio identical Hormone Replacement therapy is Human Growth Hormone. It can allow an older woman or gentleman to live a healthier, longer, and more active life. The greatest point to be made about Human Growth Hormone is that it truly is not all that fantastic of a concept. Growth Hormone is naturally produced by the body, and it is produced in large quantities when we are in our youth. The longer we can retain Growth Hormone, the more we can slow down many of the devastating effects of aging.

It makes perfect sense. Growth Hormone is vital to growth processes within the body. GH supplement is provided to young children who are maligned with slow skeletal growth, and it allows them to grow taller than they would without treatment. The problem is that Human Growth Hormone levels drop at a steady rate in all men and women after they enter adulthood. After decades of research, we now know that Human Growth Hormone provides significant benefits throughout the lifespan. For this reason, we must conclude that Growth Hormone is vital for the maintenance and rejuvenation of the body in adulthood. Research regarding the effect of Growth HRT upon elderly patients provides evidence that the main role of the hormone in adulthood is to allow one to live a longer, healthier life of higher quality.

Where does Human Growth Hormone Originate?

Human Growth Hormone is produced in the anterior area of the pituitary gland which is buried deep within the brain. Human Growth Homrone is a microscopic protein molecule. It has chemical similarities with insulin, though HGH is secreted in brief, sharp pulses after a person exercises or in the initial hours of rest. Human Growth Hormone only stays in the blood stream for a few minutes. For this reason, it in incredibly difficult to measure Human Growth Hormone in serum. Though it does not remain in the blood stream for long, HGH binds to the liver which turns some of the hormone into Somatomedin-C, which is also known well by the name Insulin-Like Growth Factor I (IGF-1) Since IGF-I stays in the blood for up to one and a half days, a blood sample that contains IGF-I proves that HGH is being produced. Normal IGF-I levels in the blood tend to appear in volumes of 200 to 450 ng/ml. However, one third of men and women that are over the age of fifty display levels of HGH that are lower than 200 ng/ml. During puberty, Human Growth Hormone levels are at their peak and IGF-I reaches levels of 600-800 ng/ml or even higher. In normal women and men under the age of forty, less than five percent have levels of HGH that are below 250 ng/ml! After the age of 40 many women and men have a level of Human Growth Hormone that is similar to that of an octogenarian! Human Growth Hormone may be the means by which we can age gracefully.

When IGF-I levels drop under the baseline adult range, energy levels and bone and muscle strength tend to decrease as well. Cell regrowth, tissue repair, vital organ repair, general rejuvenative ability, enzyme production, and hair, skin, and nail revitalization will begin to occur less effectively

Though decreased levels of Human Growth Hormone and increased age are certainly directly correlated, those who lose full function of the pituitary gland, whether through accident, infection, or surgery, will immediately begin to be maligned with acute ill effects.

It is vital to understand that natural HGH will inevitably decline with age!

In those who are utterly lacking a functional pituitary, body composition changes and a shift occurs where the amount of fat increases by 7-25%. Lean mass of the body decreases at a similar rate. Mass and strength of the muscles diminish as well. Studies in regard to bone density show that in those patients with no pituitary input, it seems as though an individual ages by around 15 years by analysis of the density of both the long bones and the spinal bones. As Human Growth Hormone flags, weight increases of as much as fifty pounds occur. Beyond this, there are issues regarding cholesterol as well. Levels of triglycerides go up, and good HDL cholesterol suffers. Cardiovascular complications occur more often, which could be related to the thickening of the vascular walls of the heart. Decreased output by the heart leads to further problems.

Hormonal insufficiencies such as these often lead to people such as these to report a serious decline in their capacity to exercise, and they often suffer from cardiovascular disease, which leads to early mortality. Also, they report issues with loneliness, depression, focus, and fatigue. In general, they just suffer from a general lack of well-being.

Hope on the Horizon: Food and Drug Administration approves Human Growth Hormone for Use by Adults

Recombinant HGH has recently gained approval by the FDA for women and men of adulthood for the therapy of Growth Hormone Deficiency and hypopituitarism. If a person suffers from hypopituitarism, Human Growth Hormone therapy is compatible under the majority of insurance policies. After being approved for therapy, patients are administered Human Growth Hormone treatment and testing under professional supervision. Our experience and the revelations of research provide ample evidence that Human Growth Hormone Replacement Therapy offers benefits to those that have insufficient levels of Growth Hormone. This deficiency is often referred to as Somatotropin Deficiency Syndrome. The Conscious Evolution Medical Institute also provides Human Growth Hormone treatment for patients who suffer from Somatomedin-C (Insulin-Like Growth Factor-1) levels under 200 ng/ml that are approved in our Internal Review Board registry, but these patients are required to pay for their treatment.

Please realize that by undergoing a global hormone replacement regimen, administering progesterone, estrogen, testosterone, thyroid, pregnenolone, or DHEA (other vital hormones which a patient may be co-deficient) when necessary in addition to Human Growth Hormone, one can minimize the costs, side-effects, and dosage of treatment.

Growth Hormone Replacement Therapy Changes

Administration of Human Growth Hormone often leads to substantial changes in the composition of the body: lean muscle mass increases as fat mass percentage is reduced. Using Human Growth Hormone at the modest dosage of 0.003 mg/kg over the course of six months was shown to improve lean muscle mass in 24 patients that had a Human Growth Hormone deficiency (1). Improvements in lean muscle mass have been associated with higher levels of protein synthesis, along with muscle function and mass. The percentage of body fat also drops after six months of Human Growth Hormone administration. The most fat is eliminated from the areas of the trunk and the stomach, rather than the legs, neck, and arms. This fact is evidence that Human Growth Hormone replacement treatment can reverse excess stomach fat associated with Human Growth Hormone deficiency, along with reducing risks of heart problems.

Heart Health and Metabolism of Cholesterols

Human Growth Hormone Therapy for adults also shows is believed to have a positive effect upon cholesterol. In new research, it has been shown that brief regimens of Human Growth Hormone Therapy led to reduced LDL (or bad) cholesterol (7) while also boosting heart function and capacity for exercise. Participants showed higher levels of oxygen intake as well as strength during cycling exercise. Human Growth Hormone has been associated with higher levels of muscle mass and stronger and longer action upon the treadmill (8). Recent research by Fazio has shown that those who have experienced heart failure have shown improvement after being given high dosage of Human Growth Hormone Therapy.

Bone Density

Among the most stunning effects of Human Growth Hormone is its effect upon the skeletal system, muscles, and tissues. It provides a significant level of rejuvenation to these systems. Skin health improves, damaged bones heal more effectively, and significant levels of improved muscular strength have been reported during Human Growth Hormone Therapy. Others have reported that signs of aging are fading from their skin. Sometimes hair regrowth occurs in male hormone replacement therapy patients as well as female patients. These changes provide evidence that testosterone, DHEA, and Human Growth Hormone are anabolic (synthesizing) hormones that significantly rehabilitate tissue of the body. Unfortunately, as we age, catabolic processes lead to faster levels of tissue breakdown. Human Growth Hormone combats the catabolic processes of aging.

The most significant way that Human Growth Hormone effects the skeletal system is through its role in enhancing the body's ability to build and repair the bones. Human Growth Hormone stimulates the proliferation of fibroblast (ligament) and osteoblast (bone) cells in the skeletal system. In recent clinical research using the powerful techniques of single photon absorptiometry and quantitative tomography, marked increases of 4% and 5% were shown in cortical and spinal bone density over the course of twelve months of therapy in adults deficient in Human Growth Hormone (4). In conclusion, it seems that the administration of Human Growth Hormone may lead to an enhancement of multiple organ systems of the body through the positive effect it has upon the skeletal system.

More examples of Human Growth Hormone's anabolic effects include muscle gain and increased appetite, increased capacity for exercise, increased capacity of the lungs, and increased would rehabilitation. Many also report that liver spots, also known as senile keratoses, begin to fade after eight weeks of Human Growth Hormone Therapy.

Side-Effects of Low Dosage Human Growth Identical Hormone Replacement Therapy.

Human Growth Hormone dosage is a vital consideration in those who suffer from Human Growth Hormone Deficiency. Overdosage of Human Growth hormone is associated wit many signs of excess Human Growth Hormone. Among these symptoms are hypertension, carpal tunnel, and excess fluid retention. However, by using more modest doses of Human Growth Hormone, it was demonstrated by Bengtsson that only mild side-effects such as slight joint pain and mold fluid retention occur. Only a single case of carpal tunnel was discovered. In each of these cases, further restriction of the Human Growth Hormone dose lead to full elimination of the side-effects. Another clinical study only used a small dose of 0.01 mg/kg, which was injected three times each week. In this study, there were zero side-effects reported (8). Many studies provide evidence that says that low dosage Human Growth Identical Hormone replacement therapy generally only causes modest side-effects, if any.

Benefits of Hormone Replacement Therapy with HGH.

Improve Body Composition: Boost muscle mass and reduce fat for a shift in body composition of as much as 20 pounds. This change can lead to higher sex drive, as well as enhanced physical well-being and self perception.

Improve Energy: Human Growth Hormone patients report higher levels of energy without the need for afternoon snacks, stimulants, caffeine, or cigarettes. This energy boost helps them become more confident in their self control and enhances their capacity for exercise.

Improve Focus: An intriguing yet untested potential benefit of Human Growth Hormone is a return of mental focus and enhancement of memory. Since Human Growth Hormone provides improvement to the intracellular and vascular systems which support the cells, it is not at all surprising that these benefits have been reported in individuals.

The Future

Many fundamental questions remain regarding the exact medical definition and preferred treatment for partial versus total Human Growth Hormone Deficiency. Also, it is not clear whether all of the realized positive effect of full Human Growth Hormone Replacement Therapy will occur in those that undergo low dosage or combination treatments. Even so, is has already been made clear that even small doses that do not result in potential side-effects sufficient level may be sufficient for some to reach their metabolic goals. We here at the Conscious Evolution Medical Institute hope that our continued clinical evaluations will help lead to more efficient usage of Human Growth Hormone, as well as help reveal more of this wondrous therapy's benefits while eliminating incidence of increased cardiovascular and general morbidity.

Works Cited

Cuneo, Salomon, and Hesp, et al. Treatment effects of Recombinant HGH upon Metabolism and Body Composition among Adults with Growth Hormone Deficiency. New England Journal of Medicine 1989; Issue 321: Pages 1797-03.

Bengtsson. Consequences of Growth Hormone Deficiency in Adults. Acta Endocrinoligica 1993; Volume 128: Pages 2-5.

Cuneo, Salomon, Wiles et al. Effects of Human Growth Hormone Treatment Among Adults Whom are Deficient. Applied Physiology Journal 1991; Volume 70: Pages 695-700.

Halloran, Tsatsoulis, Whitehouse et al. Increased Bone Density after Human Growth Hormone Therapy Upon Deficient Adults. Journal of Clinical Endocrine Metabolism 1993;Volume 76: Pages1344-48.

McGauley, Cuneo, Salomon et al. Psych. Self-perception changes due to Growth Hormone Treatment in Adults with Human Growth Hormone Deficiency. Hormone Research 1990; Volume 33(Supplement 4): Pages 52-54.

Bengtsson, Eden, Lonn et al. Therapy of Adults with Human Growth Hormone Deficiency with Recombinant Growth Hormone therapy. Journal of Clinical Endocrine Metabolism 1993;Volume 76:Pages 309-17.

Johnston, Bengtsson. Effects of Growth Hormone and its Deficiency upon Heart and Lipids. Acta Endocrinologica 1993;Volume 128(Supplement 2):Pages 69-70.

Amato, Carella, Fazio et al. Heart Structure, Metabolism of Bone, and Body Composition Heart Structure in Growth Hormone-Deficient Adults Effects of Human Growth Hormone Replacement Therapy at Mild Dosage. Journal of Clinical Endocrinology and Metabolism. 1993;Volume 77:Pages1671-76.

Fazio, Sabatini, Capaldo, et.al. Test study of GH in the treatment of cardiomyopathy. New England Journal of Medicine. 1996;Volume 334:Pages 809-14.

The above information does not provide sufficient evidence to self-diagnose a particular procedure or treatment. It should not lead one to believe that this is the only treatment option available.


HGH on 60 Minutes


Written by Dr. Welsh, Published on 28 September 2017

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IGF-1 stimulates Corticospinal Motor Neurons

ScienceDaily November 4, 2006

In a new scientific study, a growth factor known as Insulin-like Growth Factor 1 (IGF) has been shown to encourage the rapid growth of corticospinal motor neurons (CSMN). These neurons are vitally important because they connect the brain to the spinal cord, allowing the brain to interact and send signals throughout the body. Although these neurons are necessary for the brain to control the human body, these cells are highly susceptible diseases such as Lou Gehrigs disease, also known as amyotrophic lateral sclerosis.

What is ALS?

ALS is a disease in which the neurons in the corticospinal region of the brain and spinal cord atrophy and die. Over time, this leads to muscle tissue breakdown and neurological misfires by the brain. The muscles twitch as a result of incomplete connections. Eventually, ALS leads to a total breakdown of these neuron passage ways which inevitably completely paralyze the arms and legs and eventually the torso as well. ALS becomes ultimately fatal as the chest muscles lose contact and the patient is no longer able to breathe on his or her own. Strangely, ALS only effects motor movement, and has no effect on the brain or perception, although the effects on the body prove fatal over time.

IGF-1 Catalyses Axon Growth

In a recent publication of Nature Neuroscience, 2 researchers stationed at the Harvard Stem Cell Institute and the Massachusetts General Hospital have developed an accurate description of how IGF-1 leads to significant growth increases in the axons of corticospinal motor neurons in vitro testing. Axons are long, thin strands that protrude from neurons, connecting them to other cells and allowing them to send messages via electrical signal. When these axons atrophy, it reduces the ability of neurons to connect with one another and form sufficient circuits in order to efficiently deliver messages.

The Devastating Effects of CSMN Atrophy

Although in vitro testing is a long way from applicable medical theory, it is proof of concept and shows how vital IGF-1 is to the human body and how future medical breakthroughs may be able to treat patients who suffer from illnesses which endanger CSMN cells. Corticospinal motor axons are responsible for sending messages from the brain to the body in order to control the muscles. If there is a breakdown in these cells, the brain will no longer be able to control the muscles and paralysis and eventual death results.

Researchers in this study found that IGF-1 is essential for the growth of CSM axons. If motor neurons are not provided sufficient IGF-1, their axons will not grow sufficiently in vitro. In addition to this, IGF-1 has been proven vitally important in motor neuron maintenance in animal research as well. When IGF-1 production was suspended in laboratory mice, it was found that Corticospinal motor axons did not grow as long or connect to adjacent cells as efficiently.

IGF-1 Proven Vital to CSMN Axon Growth

Jeffrey Macklis is the director of the Center for Nervous System Repair at Harvard Medical School. He is also the primary author of the study. He says that the research conducted by his team proves that IGF-1 has the ability to rapidly increase both the rate at which corticospinal motor axons grow and the length at which they grow. Axons are well known for their ability to extend themselves long distances in comparison to the core cell. This study is the first piece of evidence that directly shows that IGF-1 encourages the growth of corticospinal motor neurons.

Why is this Important?

Discovering this link is encouraging to Dr. Macklis, because it adds to the body of knowledge regarding how CSMN cells function and what stimuli help them develop. In addition to that, this knowledge may lead to future breakthroughs involving either IGF-1 or Human Growth Hormone in which we may eventually be able to treat injuries of the spinal cord and neuron disorders such as ALS more effectively. Today, the range of options are quite small, but as the breadth of options increases with advances in medical knowledge, it is almost inevitable that new techniques will arise that will provide improved outcomes for these patients.

What makes Corticospinal Motor Neurons unique?

CSMN cells are unique among neuron cells within the body. Their central bodies are located in the brain, but their axons reach all the way from the brain to their target neuron in the spinal cord. Some axons extend all the way to the tip of the spinal cord, which in humans can be as long as three feet. Like most neurons of the brain and spinal cord, these cells have an incredibly limited capability to reproduce, and for this reason they are incredibly at risk when severe spinal trauma or particular medical disorders destroy their tissue.

Damage to these cells directly leads to paralysis of the corresponding areas of the body which are controlled by the particular group of Corticospinal Neurons. Although these axons are incredibly long, they have been very difficult to study until quite recently, because of the huge number of other types of neurons occupying the same space within the cerebral cortex. There are literally hundreds of kinds on neurons embedded within the tissue contained by the spinal cord. As a result, we do not know a significant amount about how CSMN cells grow and develop, although we are keenly aware of their function and the effects of their atrophy.

Isolating CSMN Cells for Study

Because of the difficulty of studying living Corticospinal Motor Neurons, Dr. Macklis and his post-doc research fellow Dr. Hande Ozdinler had to come up with an innovative way to remove CSMN cells from their natural environment and isolate them in solitary populations in vitro. Through a mixture of biological hypothesis and trial-and-error, Dr. Macklis discovered that IGF-1 was one of the primary candidates which seemed to facilitate the growth of CSMN cells.

By utilizing these isolated neurons, the duo was able to prove in a laboratory environment that it was possible to encourage the growth of Corticospinal Motor Neurons with the application of IGF-1. They proved this in two ways. One way that they achieved this growth was by directly applying IGF-1 to the purified cultures. The second way was by applying IGF-1 to the purified culture via microbeads coated in the hormone.

They found that CSMN axons grew by fifteen to twenty times in length as a direct result of IGF-1 stimulation. In a living system, this growth rate had only been observed during the initial growth of the axons. When IGF-1 was removed from the system, CSMN axons again only grew at the pace they did during the control stage. This proves that IGF-1 is central to the complete development of the Corticospinal Motor Axons.

A number of different experiments were conducted after they had made the connection between IGF-1 and CSMN development. In these experiments they tested the effect of IGF-1 on another form of neuron and discovered that it had no appreciable effect. They also tested other types of Human Growth Factor, applying them to CSMN cells in order to see if any other hormones could produce a similar effect. Only IGF-1 was able to rapidly increase the rate at which CSMN cells developed.

IGF-1 only effects the CSMN Axon

Dr. Macklis and Dr. Ozdinler were also able to show that IGF-1 did not play an active role in the survival mechanisms of the core of the cell. It appears that IGF-1 only encourages the rapid lengthening of CSMN axons rather than having any influence on the central development of the cell. The duo performed tests on living laboratory mice in which they blocked the pathway which routed IGF-1 to the spinal cord. This alteration had no effect on general cellular health but retarded the development of the CSMN axons. This animal test proved that the in vitro evidence could be applied to the biology of a living subject.

Understanding the Role of IGF-1 in Corticospinal Motor Cells

Dr. Macklis says that fully revealing IGF-1s role in the development of Corticospinal Motor Axons is vital to fostering a more complete understanding about both how CSMN cells function and how we can prevent or cure spinal disorders which prevent CSMN Neurons from performing their necessary task of muscle control.

Although it may still be a long ways off, studies such as these represent the initial steps in creating treatments which can alleviate degenerative disorders such as Lou Gehrigs disease. In addition to this, if scientists can unlock the mechanism by which the hormone IGF-1 develops CSMN axons, we may one day be able to regenerate CSMN cells utilizing a mixture of IGF-1 therapy and neuronal stem cells.

Dr. Macklis research was fostered by grants from that Harvard Center for Neurodegeneration and Repair, the ALS Association, and the National Institutes of Health.

This information was collected from information released by Massachusetts General Hospital

The New and Exciting World of Human Growth Hormone Research

We are just beginning to learn about the fascinating ways in which hormones such as Human Growth Hormone and IGF-1 maintain proper health and development. IGF-1 is an incredibly versatile hormone which is derived from Human Growth Hormone. HGH is secreted by the pituitary gland and converted into IGF-1 in the liver. After converting into IGF-1, the hormone is distributed throughout the body to perform various tasks.

We have long known that IGF-1 directly leads to the breakdown of fatty tissue, enhancing weight loss and providing energy and nutrients required for muscle development. There are many other purposes of IGF-1 which we are just now discovering, and we will likely be learning more about its fantastic physiological benefits for decades to come. IGF-1 plays a central role in anti-aging and long-term health maintenance as a result of its interaction with fat and muscle tissue. Over time and as a result of these studies, Hormone Replacement Therapy will become even more popular as the litany of benefits continues to be unearthed.

The future of IGF-1 Treatments

What Dr. Macklis research shows is that IGF-1 also plays a significant role in regenerative medicine. Eventually, IGF-1 will likely play a central role in genetic enhancement. This study is perfect proof of that. There is a significant chance that stem cells will be able to be repurposed as CSMN cells in the brain and spinal cord.

If surgical implantation is possible, IGF-1 will play a central role in restoring motor function to paralyzed patients. Imagine a world where the paralyzed can walk again. Imagine a world where diseases such as ALS are treatable, rather forces which can only be stopped, not slowed. This is the future of Anti-Aging Medicine, and HGH and IGF-1 will be key players in the race to 150.


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